Company Details

  • Jinan Meigao Biopharmaceutical Technology Co., Ltd.

  •  [Shandong,China]
  • Business Type:Manufacturer
  • Main Mark: Americas , Asia , Europe
  • Exporter:51% - 60%
  • Certs:ISO9001
  • Description:OCA,Obeticholic Acid In Stock,Obeticholic Acid Cas 459789-99-2 In Stock,Cas 459789-99-2 Obeticholic Acid
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Jinan Meigao Biopharmaceutical Technology Co., Ltd.

OCA,Obeticholic Acid In Stock,Obeticholic Acid Cas 459789-99-2 In Stock,Cas 459789-99-2 Obeticholic Acid

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Obeticholic Acid

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Obeticholic Acid , cas#459789-99-2 Obeticholic acid (abbreviated to OCA, trade name Ocaliva), is a semi-synthetic bile acid analogue which has the chemical structure 6α-ethyl-chenodeoxycholic acid. It is used as a drug to treat primary biliary...

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cas 459789-99-2 Obeticholic Acid  in stock

Obeticholic acid (abbreviated to OCA, trade name Ocaliva), is a semi-synthetic bile acid analogue which has the chemical structure 6α-ethyl-chenodeoxycholic acid. It is used as a drug to treat primary biliary cholangitis, and is undergoing...

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Obeticholic Acid (abbreviated to OCA, trade name Ocaliva,6-ECDCA)

Obeticholic acid increases the expression of FXR-regulated genes in rat hepatocytes Obeticholic acidreduces expression of liver JNK-1 and JNK-2 . Obeticholic acid (256 μg/mL) shows complete inhibition of bacterial growth in all strains tested. Intestinal permeability remains unaffected after Obeticholic acid-addition to an IFN-γ-exposed intestinal epithelium of Caco-2 cells

In Vivo Obeticholic acid (10 mg/kg/day) completely reverted cholestasis induced by E217α. Administration of 6-ECDCA partially prevents the impairment in total bile acid output caused by E217α by increasing the relative abundance of β-MCA and TCDCA and TDCA[1]. INT-747 (10 mg/kg) and HS increases the pulmonary congestion in the animals.INT-747 does not improve renal pathology in the HS-fed animals[2]. INT-747 (5 mg/kg) significantly increases survival in BDL rats. INT-747-treated BDL rats exhibits a significant selective ileal increase in expression of pore-closing claudin-1. Ileal expression of ZO-1 is significantly up-regulated in INT-747-treated BDL rats[3].



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